QT interval prolongation among patients treated with angiogenesis inhibitors

Target Oncol. 2009 Apr;4(2):89-97. doi: 10.1007/s11523-009-0111-3. Epub 2009 May 6.

Abstract

Among toxicities associated with molecular targeted agents (MTA), cardiovascular toxicities remain largely unknown or underestimated. Their frequency is variable and dependent on the compound. A high incidence of hypertension, symptomatic or asymptomatic left ventricular systolic dysfunction, acute coronary syndrome, arterial and venous thrombosis has been observed in patients receiving MTA. One of the most threatening complications of angiogenic inhibitors (AI) could be QT prolongation with the risk of torsade de pointes (TdP) and sudden death. QT prolongation and torsade de pointes accounted for 29% of cardiac and non-cardiac post-marketing withdrawals. The assessment of the effects of drugs on cardiac repolarization is the subject of recent guidelines and recommendations. Regulatory agencies now require practically every new pharmaceutical compound to undergo a thorough investigation of its propensity to modify cardiac repolarization. To reduce the incidence of QT prolongation and torsade de pointes in patients receiving AI, cancer patients should be closely monitored while receiving AI.

Publication types

  • Review

MeSH terms

  • Angiogenesis Inhibitors / administration & dosage
  • Angiogenesis Inhibitors / adverse effects*
  • Arrhythmias, Cardiac / chemically induced
  • Arrhythmias, Cardiac / therapy
  • Drug Evaluation, Preclinical
  • Electric Countershock
  • Electrocardiography / drug effects*
  • Heart / drug effects*
  • Heart / physiology
  • Humans
  • Hypertension / chemically induced*
  • Hypertension / therapy
  • Magnesium / therapeutic use
  • Protein Kinase Inhibitors / administration & dosage
  • Protein Kinase Inhibitors / adverse effects*
  • Venous Thrombosis / chemically induced
  • Venous Thrombosis / drug therapy
  • Ventricular Dysfunction / chemically induced
  • Ventricular Dysfunction / therapy
  • Withholding Treatment

Substances

  • Angiogenesis Inhibitors
  • Protein Kinase Inhibitors
  • Magnesium