Inhibition of cell proliferation and glucose uptake in human laryngeal carcinoma cells by antisense oligonucleotides against glucose transporter-1

Head Neck. 2009 Dec;31(12):1624-33. doi: 10.1002/hed.21137.

Abstract

Background: Malignant cells show increased glucose uptake in vitro and in vivo, which is thought to be mediated by glucose transporters. In this study, we investigated the effect of plasmid-derived antisense RNA against the Glut-l gene on proliferation and glucose uptake in laryngeal carcinoma Hep-2 cells.

Methods: The expression plasmids pcDNA3.1(+)-Glut-1 and pcDNA3.1(+)-anti Glut-1 were constructed. The MTT method was used to assess cell growth inhibition. The expression of Glut-1 mRNA and protein was detected by reverse transcriptase-polymerase chain reaction and Western blotting, respectively.

Results: After transfection, Glut-1 AS clearly inhibited glucose uptake and cell growth in Hep-2 cells, and we observed a decrease in the expression of Glut-1 mRNA and protein in Hep-2 cells.

Conclusions: Glut-1 AS decreases glucose uptake and inhibits the proliferation of Hep-2 cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Blotting, Western
  • Cell Line, Tumor / cytology
  • Cell Line, Tumor / drug effects
  • Cell Proliferation / drug effects*
  • Glucose / pharmacokinetics*
  • Glucose Transporter Type 1 / drug effects
  • Glucose Transporter Type 1 / metabolism*
  • Humans
  • Laryngeal Neoplasms / metabolism*
  • Laryngeal Neoplasms / pathology*
  • Oligonucleotides, Antisense / pharmacology*
  • Plasmids / drug effects
  • Plasmids / metabolism
  • Polymerase Chain Reaction
  • Probability
  • Reverse Transcriptase Polymerase Chain Reaction
  • Transfection

Substances

  • Glucose Transporter Type 1
  • Oligonucleotides, Antisense
  • Glucose