Therapeutic targeting of microenvironmental interactions in leukemia: mechanisms and approaches

Drug Resist Updat. 2009 Aug-Oct;12(4-5):103-13. doi: 10.1016/j.drup.2009.06.001. Epub 2009 Jul 25.

Abstract

In hematological malignancies, there are dynamic interactions between leukemic cells and cells of the bone marrow microenvironment. Specific niches within the bone marrow microenvironment provide a sanctuary for subpopulations of leukemic cells to evade chemotherapy-induced death and allow acquisition of a drug-resistant phenotype. This review focuses on molecular and cellular biology of the normal hematopoietic stem cell and the leukemia stem cell niche, and of the molecular pathways critical for microenvironment/leukemia interactions. The key emerging therapeutic targets include chemokine receptors (CXCR4), adhesion molecules (VLA4 and CD44), and hypoxia-related proteins HIF-1alpha and VEGF. Finally, the genetic and epigenetic abnormalities of leukemia-associated stroma will be discussed. This complex interplay provides a rationale for appropriately tailored molecular therapies targeting not only leukemic cells but also their microenvironment to ensure improved outcomes in leukemia.

Publication types

  • Review

MeSH terms

  • Antineoplastic Agents / pharmacology
  • Antineoplastic Agents / therapeutic use*
  • Apoptosis
  • Bone Marrow / drug effects*
  • Bone Marrow / metabolism
  • Bone Marrow / pathology
  • Cell Communication
  • Hematopoiesis
  • Hematopoietic Stem Cells / metabolism
  • Hematopoietic Stem Cells / pathology
  • Humans
  • Leukemia / drug therapy*
  • Leukemia / etiology
  • Leukemia / metabolism
  • Leukemia / pathology
  • Neovascularization, Pathologic / metabolism
  • Neovascularization, Pathologic / prevention & control

Substances

  • Antineoplastic Agents