Polymorphisms in thrombophilia and renin-angiotensin system pathways, preterm delivery, and evidence of placental hemorrhage

Am J Obstet Gynecol. 2009 Sep;201(3):317.e1-9. doi: 10.1016/j.ajog.2009.05.060.

Abstract

Objective: The purpose of this study was to analyze functional polymorphisms in candidate genes (methylenetetrahydrofolate reductase [MTHFR]677C>T, MTHFR1298A>C, factor 5 1691G>A [FVL], and angiotensinogen (AGT)-6G>A) in relation to a hypothesized placental hemorrhage pathway to preterm delivery (PTD).

Study design: We assessed maternal genotypes, pregnancy outcomes, and placental pathologic evidence among 560 white and 399 black women who were recruited at mid trimester into a prospective cohort study (1998-2004). Odds of dominant genotypes were calculated for PTDs with (n = 56) or without (n = 177) evidence of placental hemorrhage (referent = term) with the use of race-stratified polytomous logistic regression models.

Results: Among white women, FVL GA/AA and AGT(-6) GA/AA were both associated with hemorrhage-related PTDs (odds ratio [OR], 4.8; 95% confidence interval [CI], 1.6-14.2 and OR, 3.8; 95% CI, 1.3-10.5, respectively), but not other PTDs (ORs, 1.2 and 0.9, respectively). FVL GA/AA was associated with placental abruption (OR, 5.8; 95% CI, 1.1-30) among white women. All results were null for MTHFR genotypes.

Conclusion: FVL and AGT variant genotypes were associated specifically with hemorrhage-related PTDs.

Publication types

  • Multicenter Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adult
  • Angiotensinogen / genetics*
  • Factor V / genetics*
  • Female
  • Gene Frequency
  • Genotype
  • Hemorrhage / genetics
  • Humans
  • Logistic Models
  • Methylenetetrahydrofolate Reductase (NADPH2) / genetics*
  • Placenta Diseases / genetics
  • Point Mutation / genetics
  • Polymorphism, Genetic
  • Pregnancy
  • Pregnancy Outcome
  • Premature Birth / genetics*
  • Renin-Angiotensin System / genetics*
  • Thrombophilia / genetics
  • Young Adult

Substances

  • factor V Leiden
  • Angiotensinogen
  • Factor V
  • Methylenetetrahydrofolate Reductase (NADPH2)