CpG ODN pretreatment attenuates concanavalin A-induced hepatitis in mice

Int Immunopharmacol. 2010 Jan;10(1):79-85. doi: 10.1016/j.intimp.2009.09.025. Epub 2009 Oct 7.

Abstract

T cell-mediated hepatic damage plays a key role in the pathogenesis of liver diseases such as autoimmune hepatitis, viral hepatitis and acute liver failure. CpG-containing oligodeoxynucleotides (CpG ODN), a ligand for toll-like receptor (TLR) 9, is widely used as an immunological adjuvant. In the present study, we investigated the effect of CpG ODN on T cell-mediated liver injury in a murine model of concanavalin A (Con A)-induced hepatitis. We found that the aminotransferase level was significantly decreased in CpG ODN pretreated mice and the survival of the mice was markedly prolonged. CpG ODN pretreatment inhibited NF-kappaB DNA binding activity. As a result, the systemic/liver levels of TNF-alpha and IFN-gamma were significantly suppressed. Furthermore, the activation of inflammatory cells was diminished by CpG ODN pretreatment. These results suggest that CpG ODN pretreatment protects the mice from Con A-induced liver injury via inhibiting hepatocyte apoptosis, inflammation and activation of lymphocytes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Line
  • Cell Separation
  • Chemical and Drug Induced Liver Injury / drug therapy*
  • Chemical and Drug Induced Liver Injury / immunology
  • Concanavalin A / toxicity
  • Drug Antagonism
  • Flow Cytometry
  • Hepatitis / drug therapy*
  • Hepatitis / immunology
  • Interferon-gamma / genetics
  • Interferon-gamma / metabolism*
  • Liver / drug effects*
  • Liver / immunology
  • Liver / metabolism
  • Liver / pathology
  • Lymphocyte Activation / drug effects
  • Macrophages / drug effects
  • Macrophages / metabolism
  • Macrophages / pathology
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mitogens / toxicity
  • NF-kappa B / immunology
  • NF-kappa B / metabolism*
  • Oligodeoxyribonucleotides, Antisense / administration & dosage
  • Transcriptional Activation / drug effects
  • Tumor Necrosis Factor-alpha / genetics
  • Tumor Necrosis Factor-alpha / metabolism*

Substances

  • Mitogens
  • NF-kappa B
  • Oligodeoxyribonucleotides, Antisense
  • Tumor Necrosis Factor-alpha
  • Concanavalin A
  • Interferon-gamma