Conditional knockdown of Nanog induces apoptotic cell death in mouse migrating primordial germ cells

Development. 2009 Dec;136(23):4011-20. doi: 10.1242/dev.041160.

Abstract

The pluripotency factor Nanog is expressed in peri-implantation embryos and primordial germ cells (PGCs). Nanog-deficient mouse embryos die soon after implantation. To explore the function of Nanog in germ cells, Nanog RNA was conditionally knocked down in vivo by shRNA. Nanog shRNA transgenic (NRi-Tg) mice were generated through the formation of germline chimeras with NRi-Tg embryonic stem cells. In E12.5 Cre-induced ER-Cre/NRi-Tg and TNAP-Cre/NRi-Tg double-transgenic embryos, the number of alkaline phosphatase-positive and SSEA1-positive PGCs decreased significantly. In the E9.5 and E10.5 migrating Nanog-knockdown PGCs, TUNEL-positive apoptotic cell death became prominent in vivo and in vitro, despite Oct4 expression. Single-cell microarray analysis of E10.5 Nanog-knockdown PGCs revealed significant up- and downregulation of a substantial number of genes, including Tial1, Id1 and Suz12. These data suggest that Nanog plays a key role in the proliferation and survival of migrating PGCs as a safeguard of the PGC-specific molecular network.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis / genetics*
  • Cell Death / genetics
  • Cell Movement / genetics
  • Cell Movement / physiology
  • Cell Proliferation
  • Cells, Cultured
  • Embryo, Mammalian
  • Embryonic Stem Cells / cytology
  • Embryonic Stem Cells / metabolism*
  • Genetic Vectors
  • Germ Cells / cytology
  • Germ Cells / metabolism
  • Germ Cells / physiology*
  • Homeodomain Proteins / genetics*
  • Homeodomain Proteins / metabolism*
  • Immunohistochemistry
  • In Situ Nick-End Labeling
  • Integrases / genetics
  • Integrases / metabolism
  • Lentivirus / genetics
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • Nanog Homeobox Protein

Substances

  • Homeodomain Proteins
  • Nanog Homeobox Protein
  • Nanog protein, mouse
  • Cre recombinase
  • Integrases