How the noninflammasome NLRs function in the innate immune system

Science. 2010 Jan 15;327(5963):286-90. doi: 10.1126/science.1184004.

Abstract

NLR (nucleotide-binding domain, leucine-rich repeat-containing) proteins have rapidly emerged as central regulators of immunity and inflammation with demonstrated relevance to human diseases. Much attention has focused on the ability of several NLRs to activate the inflammasome complex and drive proteolytic processing of inflammatory cytokines; however, NLRs also regulate important inflammasome-independent functions in the immune system. We discuss several of these functions, including the regulation of canonical and noncanonical NF-kappaB activation, mitogen-activated protein kinase activation, cytokine and chemokine production, antimicrobial reactive oxygen species production, type I interferon production, and ribonuclease L activity. We also explore the mechanistic basis of these functions and describe current challenges in the field.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Cytokines / metabolism
  • Endoribonucleases / metabolism
  • Humans
  • Immunity, Innate*
  • Inflammation
  • Interferon Type I / metabolism
  • Intracellular Signaling Peptides and Proteins / chemistry
  • Intracellular Signaling Peptides and Proteins / metabolism*
  • Mitochondrial Proteins / metabolism
  • Mitogen-Activated Protein Kinases / metabolism
  • NF-kappa B / metabolism
  • Nod1 Signaling Adaptor Protein / metabolism
  • Nod2 Signaling Adaptor Protein / metabolism
  • Protein Interaction Domains and Motifs
  • Receptors, Pattern Recognition / chemistry
  • Receptors, Pattern Recognition / metabolism*
  • Signal Transduction*

Substances

  • Cytokines
  • Interferon Type I
  • Intracellular Signaling Peptides and Proteins
  • Mitochondrial Proteins
  • NF-kappa B
  • Nod1 Signaling Adaptor Protein
  • Nod2 Signaling Adaptor Protein
  • Receptors, Pattern Recognition
  • Mitogen-Activated Protein Kinases
  • Endoribonucleases
  • 2-5A-dependent ribonuclease