Inhibition of calcium mobilization is an early event in opiate-induced immunosuppression

FASEB J. 1991 May;5(8):2194-9. doi: 10.1096/fasebj.5.8.2022315.

Abstract

Morphine administered as a subcutaneous implant inhibits the initial increase in cytoplasmic free-calcium [Ca2+]i induced by mitogens in mouse splenocytes. This effect was not reproduced by incubation of splenocytes with morphine (10(-8)-10(-4) M). Analysis of splenocyte subpopulations demonstrates that this effect was manifest in both B and T cells. However, within T cell subpopulations, CD4+ but not CD8+ cells were affected. Adrenalectomy abolished this effect of morphine in CD4+ T but not CD4-, CD8- spleen cells (most likely Thy 1.2- B cells). Moreover, simultaneous administration of the opiate antagonist naltrexone blocked the effect of morphine in CD4-, CD8- spleen cells, but not in CD4+ T cells. These data indicate that the effects of morphine on mitogen-stimulated increase in [Ca2+]i may be mediated through distinct glucocorticoid-dependent and -independent mechanisms. The morphine-induced inhibition of an increase in [Ca2+]i in immune cells reported here may be an early event mediating opiate-induced immunosuppression.

MeSH terms

  • Animals
  • Biological Transport, Active / drug effects
  • Calcium / pharmacokinetics*
  • Concanavalin A / pharmacology
  • Dexamethasone / pharmacology
  • Flow Cytometry
  • Immune Tolerance / drug effects*
  • Mice
  • Mice, Inbred C57BL
  • Morphine / pharmacology
  • Naltrexone / pharmacology
  • Narcotics / pharmacology*
  • Spleen / drug effects
  • Spleen / immunology
  • Spleen / metabolism
  • T-Lymphocytes / drug effects
  • T-Lymphocytes / immunology

Substances

  • Narcotics
  • Concanavalin A
  • Naltrexone
  • Morphine
  • Dexamethasone
  • Calcium