The I allele of the angiotensin converting enzyme I/D polymorphism confers protection against coronary artery disease

Coron Artery Dis. 2010 May;21(3):151-6. doi: 10.1097/MCA.0b013e328335a042.

Abstract

Background: Mutations in genes regulating lipid metabolism, vasoactivity, and coagulation are important modulators of coronary artery disease (CAD).

Objective: This study investigated the association between allelic variants of the angiotensin converting enzyme (ACE), methytetrahydrofolate reductase, plasminogen activator inhibitor-1 and factor V genes and CAD.

Methods: Clinical, biochemical, and angiographic information were collected from 300 patients who underwent cardiac catheterization and their DNA was genotyped by restriction fragment length polymorphism.

Results: The frequency of the D allele of the ACE gene was significantly higher than the I allele in patients with more than 70% stenosis in any vessel. Among patients with more than 70% stenosis, carriers of the D allele were 2.8 times more likely to be males. The presence of the ACE I allele was negatively associated with CAD with (P=0.02 ,OR=0.38.)

Conclusion: This study describes a protective role of the ACE I allele in individuals who may be at risk of developing CAD.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Chi-Square Distribution
  • Coronary Angiography
  • Coronary Stenosis / diagnostic imaging
  • Coronary Stenosis / enzymology
  • Coronary Stenosis / genetics*
  • Coronary Stenosis / prevention & control*
  • Factor V / genetics
  • Female
  • Gene Frequency
  • Genetic Predisposition to Disease
  • Humans
  • Logistic Models
  • Male
  • Methylenetetrahydrofolate Reductase (NADPH2) / genetics
  • Middle Aged
  • Odds Ratio
  • Peptidyl-Dipeptidase A / genetics*
  • Phenotype
  • Plasminogen Activator Inhibitor 1 / genetics
  • Polymorphism, Genetic*
  • Risk Assessment
  • Risk Factors
  • Severity of Illness Index

Substances

  • Plasminogen Activator Inhibitor 1
  • SERPINE1 protein, human
  • factor V Leiden
  • Factor V
  • Methylenetetrahydrofolate Reductase (NADPH2)
  • Peptidyl-Dipeptidase A