[Autoimmune epilepsy]

Rev Med Suisse. 2010 May 5;6(247):925-9.
[Article in French]

Abstract

There is increasing recognition of an autoimmune origin of pharmacoresistant epileptic disorders. Besides the paraneoplastic limbic encephalopathies (LE), reports of syndromes of non-paraneoplastic LE are increasingly reported in the last 5-10 years. Three antibodies are now relatively well described: Voltage-gated potassium channels (VGKC), glutamic acid decarboxylase (GAD) and N-methyl-D-aspartate receptor-(NMDA) antibodies. We review clinical syndromes, associated imaging and laboratory findings. While most reports arise from adult populations, children and adolescents are also concerned as evidenced by increasing observations. Early recognition is mandatory, since early immunomodulatory treatment appears to be related to significantly better outcome.

Publication types

  • English Abstract
  • Review

MeSH terms

  • Autoantibodies / blood*
  • Autoantibodies / immunology
  • Biomarkers / blood
  • Epilepsy / blood
  • Epilepsy / diagnosis
  • Epilepsy / drug therapy
  • Epilepsy / immunology*
  • Glutamate Decarboxylase / blood
  • Humans
  • Immunologic Factors / therapeutic use
  • Limbic Encephalitis / complications*
  • Limbic Encephalitis / immunology
  • N-Methylaspartate / blood
  • Neuropsychological Tests
  • Paraneoplastic Syndromes / blood
  • Paraneoplastic Syndromes / diagnosis
  • Paraneoplastic Syndromes / drug therapy
  • Paraneoplastic Syndromes / immunology*
  • Potassium Channels, Voltage-Gated / blood
  • Treatment Outcome

Substances

  • Autoantibodies
  • Biomarkers
  • Immunologic Factors
  • Potassium Channels, Voltage-Gated
  • N-Methylaspartate
  • Glutamate Decarboxylase