Ca2(+)-dependence of endothelin-1 induced contraction of rat tail artery

Gen Pharmacol. 1991;22(2):399-402. doi: 10.1016/0306-3623(91)90471-h.

Abstract

1. In rat tail artery, endothelin-1(ET-1) caused an increase in the cytosolic free Ca2+ level ([Ca2+]i) followed by a relatively sustained but not steady-state contraction in Ca2+ containing solution. 2. In the early phase of the contraction, the rate of increase in [Ca2+]i, was much faster than that in muscle tension. However, after the increases in [Ca2+]i and muscle tension reached at their peaks, there was a good correlation between the changes of the two parameters. 3. ET-1 could not induce an apparent contractile response in Ca2(+)-free medium, notwithstanding it evoked a [Ca2+]i transient in this medium. 4. The results indicate that ET-1 induce a contraction of rat tail artery which is almost fully dependent on the [Ca2+]i changes, and may inhibit the Ca2(+)-sensitivity of the contractile filaments in the early phase of the contraction.

MeSH terms

  • Animals
  • Arteries / drug effects
  • Caffeine / pharmacology
  • Calcium / metabolism
  • Calcium / physiology*
  • Endothelins / pharmacology*
  • Female
  • Fura-2 / pharmacology
  • In Vitro Techniques
  • Muscle Contraction / drug effects
  • Muscle, Smooth, Vascular / drug effects*
  • Rats
  • Rats, Inbred Strains
  • Regional Blood Flow / drug effects
  • Tail / blood supply

Substances

  • Endothelins
  • Caffeine
  • Calcium
  • Fura-2