Abstract
Thalamic neurochemical abnormalities may underlie psychotic symptoms and auditory event-related potential (ERP) abnormalities in schizophrenia. We investigated this hypothesis in subjects at risk of psychosis using magnetic resonance spectroscopy and electroencephalography (EEG). Reduced thalamic glutamate plus glutamine and N-acetyl aspartate levels were associated with abnormal frontal ERPs, supporting a thalamic basis for filtering impairments.
Copyright 2010 Elsevier Ireland Ltd. All rights reserved.
MeSH terms
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Acoustic Stimulation / methods
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Adult
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Aspartic Acid / analogs & derivatives
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Aspartic Acid / metabolism
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Electroencephalography
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Evoked Potentials, Auditory / physiology*
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Female
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Frontal Lobe / physiopathology
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Glutamic Acid / metabolism
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Glutamine / metabolism
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Humans
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Linear Models
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Magnetic Resonance Spectroscopy / methods
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Male
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Neurochemistry*
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Protons
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Radionuclide Imaging
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Schizophrenia / diagnostic imaging
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Schizophrenia / pathology*
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Schizophrenia / physiopathology
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Thalamus / metabolism*
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Young Adult
Substances
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Protons
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Glutamine
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Aspartic Acid
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Glutamic Acid
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N-acetylaspartate