Reexpression of HPRT activity following cell fusion with polyethylene glycol

Biochem Genet. 1978 Apr;16(3-4):227-37. doi: 10.1007/BF00484080.

Abstract

Polyethylene glycol-1000 (PEG-1000) induced fusion of HPRT (E.C. 2.4.2.8) deficient Chinese hamster cells with alpha-galactosidase A (E.C. 2.3.1.22) deficient cells from a patient with Fabry's disease yielded hybrids which contained both human and hamster HPRT, G6PD (E.C. 1.1.1.49), and APRT (E.C. 2.4.2.7) and Chinese hamster alpha-galactosidase B. Thus PEG-1000 mediated somatic cell fusion led to reexpression of Chinese hamster HPRT. It did not restore the expression of human alpha-galactosidase. Since PEG-1000 treatment of HPRT- Chinese hamster cells in the absence of human cells yielded no HPRT+ cells, it is concluded that the element responsible for the restoration of rodent HPRT was contributed by the human cells and not by the agent employed to promote fusion.

Publication types

  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adenine Phosphoribosyltransferase / genetics
  • Animals
  • Cell Fusion / drug effects*
  • Cricetinae
  • Genes
  • Glucosephosphate Dehydrogenase / genetics
  • Humans
  • Hybrid Cells / enzymology*
  • Hypoxanthine Phosphoribosyltransferase / biosynthesis
  • Hypoxanthine Phosphoribosyltransferase / genetics*
  • Isoenzymes / genetics
  • Polyethylene Glycols / pharmacology*
  • alpha-Galactosidase / genetics

Substances

  • Isoenzymes
  • Polyethylene Glycols
  • Glucosephosphate Dehydrogenase
  • Adenine Phosphoribosyltransferase
  • Hypoxanthine Phosphoribosyltransferase
  • alpha-Galactosidase