Abstract
The development of a new series of p38α inhibitors resulted in the identification of two clinical candidates, one of which was advanced into a phase 2 clinical study for rheumatoid arthritis. The original lead, an lck inhibitor that also potently inhibited p38α, was a screening hit from our kinase inhibitor library. This manuscript describes the optimization of the lead to p38-selective examples with good pharmacokinetic properties.
MeSH terms
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Administration, Oral
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Arthritis, Rheumatoid / drug therapy
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Biological Availability
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Cell Line
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Clinical Trials as Topic
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Drug Discovery / methods*
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Humans
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Mitogen-Activated Protein Kinase 14 / antagonists & inhibitors*
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Mitogen-Activated Protein Kinase 14 / chemistry
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Models, Molecular
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Protein Conformation
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Protein Kinase Inhibitors / administration & dosage
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Protein Kinase Inhibitors / chemistry
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Protein Kinase Inhibitors / pharmacokinetics
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Protein Kinase Inhibitors / pharmacology
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Pyridones / administration & dosage
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Pyridones / chemistry
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Pyridones / pharmacokinetics*
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Pyridones / pharmacology*
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Pyrimidines / administration & dosage
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Pyrimidines / chemistry
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Pyrimidines / pharmacokinetics*
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Pyrimidines / pharmacology*
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Structure-Activity Relationship
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Substrate Specificity
Substances
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6-(2,4-difluorophenoxy)-8-methyl-2-(tetrahydro-2H-pyran-4-ylamino)pyrido(2,3-d)pyrimidin-7(8H)-one
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Protein Kinase Inhibitors
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Pyridones
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Pyrimidines
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pamapimod
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Mitogen-Activated Protein Kinase 14