Abnormal thyroid function tests in children on ethionamide treatment

Int J Tuberc Lung Dis. 2011 Sep;15(9):1191-3, i. doi: 10.5588/ijtld.10.0707.

Abstract

Ethionamide (ETH) treatment may cause hypothyroidism. Clinical data, serum thyroid stimulating hormone (TSH) and free thyroxine (fT4) levels were retrospectively assessed in 137 children receiving anti-tuberculosis treatment including ETH. Abnormal thyroid function tests (TFTs) were recorded in 79 (58%) children: elevated serum TSH and suppressed fT4 (n = 30), isolated elevated serum TSH (n = 20), isolated low serum fT4 (n = 28) and isolated low TSH (n = 1). The risk for biochemical hypothyroidism was higher for children on regimens including para-aminosalicylic acid and in human immunodeficiency virus infected children. TFT abnormalities are frequent in children on ETH and are mainly due to primary hypothyroidism or euthyroid sick syndrome.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Aminosalicylic Acid / adverse effects
  • Aminosalicylic Acid / therapeutic use
  • Antitubercular Agents / adverse effects*
  • Antitubercular Agents / therapeutic use
  • Child
  • Child, Preschool
  • Ethionamide / adverse effects*
  • Ethionamide / therapeutic use
  • Euthyroid Sick Syndromes / chemically induced*
  • Female
  • HIV Infections / epidemiology
  • Humans
  • Hypothyroidism / chemically induced*
  • Infant
  • Male
  • Retrospective Studies
  • Thyroid Function Tests
  • Thyrotropin / blood
  • Thyroxine / blood
  • Tuberculosis / drug therapy

Substances

  • Antitubercular Agents
  • Aminosalicylic Acid
  • Thyrotropin
  • Ethionamide
  • Thyroxine