Epigenetic regulation of osteoclast differentiation

Ann N Y Acad Sci. 2011 Dec:1240:7-13. doi: 10.1111/j.1749-6632.2011.06245.x.

Abstract

Recent studies have uncovered that epigenetic regulation, such as histone methylation and acetylation, plays a critical role in determining cell fate. In particular, the expression of key developmental genes tends to be regulated by trimethylation of histone H3 lysine 4 (H3K4me3) and lysine 27 (H3K27me3). Osteoclasts are primary cells for bone resorption, and their differentiation is tightly regulated by the receptor activator of nuclear factor κB ligand (RANKL) and a transcription factor nuclear factor-activated T cell (NFAT) c1. We found that RANKL-induced NFATc1 expression is associated with the demethylation of H3K27me3. Jumonji domain containing-3, a H3K27 demethylase, is induced in bone marrow-derived macrophages in response to RANKL stimulation and may play a critical role in the demethylation of H3K27me3 in the Nfatc1 gene.

Publication types

  • Review

MeSH terms

  • Animals
  • Bone Marrow Cells / cytology
  • Bone Marrow Cells / metabolism*
  • Bone Resorption / metabolism*
  • Cell Differentiation / physiology*
  • Epigenesis, Genetic / physiology*
  • Histones / metabolism
  • Humans
  • Jumonji Domain-Containing Histone Demethylases / metabolism
  • Methylation
  • NFATC Transcription Factors / biosynthesis
  • Osteoclasts / cytology
  • Osteoclasts / metabolism*
  • RANK Ligand / metabolism

Substances

  • Histones
  • NFATC Transcription Factors
  • NFATC1 protein, human
  • RANK Ligand
  • TNFSF11 protein, human
  • Jumonji Domain-Containing Histone Demethylases