Lyso-GM2 ganglioside: a possible biomarker of Tay-Sachs disease and Sandhoff disease

PLoS One. 2011;6(12):e29074. doi: 10.1371/journal.pone.0029074. Epub 2011 Dec 20.

Abstract

To find a new biomarker of Tay-Sachs disease and Sandhoff disease. The lyso-GM2 ganglioside (lyso-GM2) levels in the brain and plasma in Sandhoff mice were measured by means of high performance liquid chromatography and the effect of a modified hexosaminidase (Hex) B exhibiting Hex A-like activity was examined. Then, the lyso-GM2 concentrations in human plasma samples were determined. The lyso-GM2 levels in the brain and plasma in Sandhoff mice were apparently increased compared with those in wild-type mice, and they decreased on intracerebroventricular administration of the modified Hex B. The lyso-GM2 levels in plasma of patients with Tay-Sachs disease and Sandhoff disease were increased, and the increase in lyso-GM2 was associated with a decrease in Hex A activity. Lyso-GM2 is expected to be a potential biomarker of Tay-Sachs disease and Sandhoff disease.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Animals
  • Biomarkers / blood
  • Biomarkers / metabolism
  • Brain / metabolism
  • G(M2) Activator Protein / deficiency
  • G(M2) Ganglioside / analogs & derivatives*
  • G(M2) Ganglioside / blood
  • G(M2) Ganglioside / metabolism
  • Hexosaminidases / blood
  • Humans
  • Infant
  • Mice
  • Sandhoff Disease / blood
  • Sandhoff Disease / enzymology
  • Sandhoff Disease / metabolism*
  • Tay-Sachs Disease / blood
  • Tay-Sachs Disease / enzymology
  • Tay-Sachs Disease / metabolism*

Substances

  • Biomarkers
  • G(M2) Activator Protein
  • lysoganglioside G(M2)
  • G(M2) Ganglioside
  • Hexosaminidases