Anti-CD45RB/anti-TIM-1-induced tolerance requires regulatory B cells

Am J Transplant. 2012 Aug;12(8):2072-8. doi: 10.1111/j.1600-6143.2012.04055.x. Epub 2012 Apr 11.

Abstract

The role of B cells in transplant tolerance remains unclear. Although B-cell depletion often prolongs graft survival, sometimes it results in more rapid rejection, suggesting that B cells may have regulatory activity. We previously demonstrated that tolerance induction by anti-CD45RB antibody requires recipient B cells. Here, we show that anti-CD45RB in combination with anti-TIM-1 antibody has a synergistic effect, inducing tolerance in all recipients in a mouse islet allograft model. This effect depends on the presence of recipient B cells, requires B-cell IL-10 activity, and is antigen-specific. These data suggest the existence of a regulatory B-cell population that promotes tolerance via an IL-10-dependent pathway.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • B-Lymphocytes / immunology*
  • Hepatitis A Virus Cellular Receptor 1
  • Immune Tolerance*
  • Leukocyte Common Antigens / immunology*
  • Membrane Proteins / physiology*
  • Mice
  • Mice, Inbred Strains

Substances

  • Havcr1 protein, mouse
  • Hepatitis A Virus Cellular Receptor 1
  • Membrane Proteins
  • Leukocyte Common Antigens