Male gender promotes an increased inflammatory response to lipopolysaccharide in umbilical vein blood

J Matern Fetal Neonatal Med. 2012 Nov;25(11):2470-4. doi: 10.3109/14767058.2012.684165. Epub 2012 May 17.

Abstract

Objectives: To establish gender-specific differences in maternal and fetal immune response in healthy human fetuses at term.

Methods: Forty-five women with elective caesarean sections for uncomplicated singleton pregnancies were recruited for two studies. Using a multiplex biomarker immunoassay system, unstimulated maternal and fetal plasma concentrations of interleukin (IL)-1β, IL-1ra, IL-6, IL-8, macrophage inflammatory protein (MIP)-1α, and tumor necrosis factor (TNF)-α were measured from one study population. Lipopolysaccharide (LPS)-stimulated cytokine response was measured in a second study.

Results: There were no significant gender differences in either maternal or fetal unstimulated plasma cytokine concentrations, but concentrations of the proinflammatory cytokines IL-1β and IL-6 were significantly greater in male fetal LPS-stimulated samples than in female fetal samples.

Conclusions: Blood of male fetuses mounts a larger pro-inflammatory response to lipopolysaccharide (LPS). This heightened response could be a critical pathway in promoting premature rupture of membranes (PPROM) and may be associated with life long differential gender response to infection.

Publication types

  • Comparative Study
  • Evaluation Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Cytokines / blood
  • Female
  • Fetal Blood / drug effects*
  • Fetal Blood / immunology
  • Fetal Blood / metabolism
  • Humans
  • In Vitro Techniques
  • Infant, Newborn
  • Inflammation / blood*
  • Inflammation / chemically induced*
  • Lipopolysaccharides / pharmacology*
  • Male
  • Mothers
  • Osmolar Concentration
  • Pregnancy / blood
  • Sex Characteristics*
  • Umbilical Veins / chemistry
  • Umbilical Veins / drug effects
  • Umbilical Veins / immunology
  • Umbilical Veins / metabolism

Substances

  • Cytokines
  • Lipopolysaccharides