Differential DNA methylation patterns between high and low responders to a weight loss intervention in overweight or obese adolescents: the EVASYON study

FASEB J. 2013 Jun;27(6):2504-12. doi: 10.1096/fj.12-215566. Epub 2013 Mar 8.

Abstract

In recent years, epigenetic markers emerged as a new tool to understand the influence of lifestyle factors on obesity phenotypes. Adolescence is considered an important epigenetic window over a human's lifetime. The objective of this work was to explore baseline changes in DNA methylation that could be associated with a better weight loss response after a multidisciplinary intervention program in Spanish obese or overweight adolescents. Overweight or obese adolescents (n=107) undergoing 10 wk of a multidisciplinary intervention for weight loss were assigned as high or low responders to the treatment. A methylation microarray was performed to search for baseline epigenetic differences between the 2 groups (12 subjects/group), and MALDI-TOF mass spectrometry was used to validate (n=107) relevant CpG sites and surrounding regions. After validation, 5 regions located in or near AQP9, DUSP22, HIPK3, TNNT1, and TNNI3 genes showed differential methylation levels between high and low responders to the multidisciplinary weight loss intervention. Moreover, a calculated methylation score was significantly associated with changes in weight, BMI-SDS, and body fat mass loss after the treatment. In summary, we have identified 5 DNA regions that are differentially methylated depending on weight loss response. These methylation changes may help to better understand the weight loss response in obese adolescents.

Keywords: biomarkers; dieting response; epigenetics.

Publication types

  • Clinical Trial
  • Multicenter Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Aquaporins / genetics
  • DNA Methylation*
  • Dual-Specificity Phosphatases / genetics
  • Female
  • Humans
  • Intracellular Signaling Peptides and Proteins / genetics
  • Male
  • Mitogen-Activated Protein Kinase Phosphatases / genetics
  • Obesity / genetics*
  • Obesity / metabolism
  • Obesity / therapy*
  • Overweight / genetics*
  • Overweight / metabolism
  • Overweight / therapy*
  • Protein Serine-Threonine Kinases / genetics
  • Spain
  • Troponin I / genetics
  • Troponin T / genetics
  • Weight Loss / genetics*
  • Weight Loss / physiology
  • Weight Reduction Programs*

Substances

  • AQP9 protein, human
  • Aquaporins
  • Intracellular Signaling Peptides and Proteins
  • Troponin I
  • Troponin T
  • HIPK3 protein, human
  • Protein Serine-Threonine Kinases
  • Mitogen-Activated Protein Kinase Phosphatases
  • DUSP22 protein, human
  • Dual-Specificity Phosphatases