Overexpression of the urokinase receptor splice variant uPAR-del4/5 in breast cancer cells affects cell adhesion and invasion in a dose-dependent manner and modulates transcription of tumor-associated genes

Biol Chem. 2012 Dec;393(12):1449-55. doi: 10.1515/hsz-2012-0206.

Abstract

mRNA levels of the urokinase receptor splice variant uPAR-del4/5 are associated with prognosis in breast cancer. Its overexpression in cancer cells affects tumor biologically relevant processes. In the present study, individual breast cancer cell clones displaying low vs. high uPAR-del4/5 expression were analyzed demonstrating that uPAR-del4/5 leads to reduced cell adhesion and invasion in a dose-dependent manner. Additionally,matrix metalloproteinase-9 (MMP-9) was found to be strongly upregulated in uPAR-del4/5 overexpressing compared to vector control cells. uPAR-del4/5 may thus play an important role in the regulation of the extracellular proteolytic network and, by this, influence the metastatic potential of breast cancer cells.

MeSH terms

  • Breast / cytology
  • Breast / metabolism*
  • Breast / pathology
  • Breast Neoplasms / genetics*
  • Breast Neoplasms / pathology
  • Cell Adhesion
  • Cell Line, Tumor
  • Female
  • Gene Expression Regulation, Neoplastic
  • Humans
  • Matrix Metalloproteinase 9 / genetics
  • Neoplasm Invasiveness / genetics
  • RNA Splicing*
  • Receptors, Urokinase Plasminogen Activator / genetics*
  • Up-Regulation

Substances

  • Receptors, Urokinase Plasminogen Activator
  • Matrix Metalloproteinase 9