Combining fragment homology modeling with molecular dynamics aims at prediction of Ca²⁺ binding sites in CaBPs

J Comput Aided Mol Des. 2013 Aug;27(8):697-705. doi: 10.1007/s10822-013-9668-0. Epub 2013 Aug 10.

Abstract

The family of calcium-binding proteins (CaBPs) consists of dozens of members and contributes to all aspects of the cell's function, from homeostasis to learning and memory. However, the Ca²⁺-binding mechanism is still unclear for most of CaBPs. To identify the Ca²⁺-binding sites of CaBPs, this study presented a computational approach which combined the fragment homology modeling with molecular dynamics simulation. For validation, we performed a two-step strategy as follows: first, the approach is used to identify the Ca²⁺-binding sites of CaBPs, which have the EF-hand Ca²⁺-binding site and the detailed binding mechanism. To accomplish this, eighteen crystal structures of CaBPs with 49 Ca²⁺-binding sites are selected to be analyzed including calmodulin. The computational method identified 43 from 49 Ca²⁺-binding sites. Second, we performed the approach to large-conductance Ca²⁺-activated K⁺ (BK) channels which don't have clear Ca²⁺-binding mechanism. The simulated results are consistent with the experimental data. The computational approach may shed some light on the identification of Ca²⁺-binding sites in CaBPs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Binding Sites
  • Calbindins / chemistry
  • Calbindins / metabolism
  • Calcium / metabolism*
  • Calcium-Binding Proteins / chemistry
  • Calcium-Binding Proteins / metabolism*
  • EF Hand Motifs
  • Humans
  • Molecular Dynamics Simulation*
  • Molecular Sequence Data
  • RNA-Binding Proteins / chemistry
  • RNA-Binding Proteins / metabolism
  • Rats

Substances

  • Calbindins
  • Calcium-Binding Proteins
  • RNA-Binding Proteins
  • high density lipoprotein binding protein
  • Calcium