Reevaluating the role of the hippocampus in delay eyeblink conditioning

PLoS One. 2013 Aug 9;8(8):e71249. doi: 10.1371/journal.pone.0071249. eCollection 2013.

Abstract

The role of the hippocampus in delay eyeblink conditioning (DEC) remains controversial. Here, we investigated the involvement of the hippocampus in DEC with a soft tone as the conditioned stimulus (CS) by using electrolytic lesions or muscimol inactivation of guinea pig dorsal hippocampus. Interestingly, when a soft tone was used as a CS, electrolytic lesions of the hippocampus significantly retarded acquisition of the conditioned response (CR), and muscimol infusions into hippocampus distinctly inhibited the acquisition and expression of CR, but had no significant effect on consolidation of well-learned CR. In contrast, both electrolytic lesions and muscimol inactivation of hippocampus produced no significant deficits in the CR when a loud tone was used as the CS. These results demonstrate that the hippocampus is essential for the DEC when the delay task was rendered more difficult.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Behavior, Animal
  • Blinking / drug effects
  • Blinking / physiology*
  • Conditioning, Classical / drug effects
  • Conditioning, Classical / physiology*
  • Conditioning, Eyelid / drug effects
  • Conditioning, Eyelid / physiology*
  • Female
  • GABA Agonists / pharmacology
  • Guinea Pigs
  • Hippocampus / drug effects
  • Hippocampus / physiology*
  • Muscimol / pharmacology
  • Pattern Recognition, Physiological
  • Stereotaxic Techniques

Substances

  • GABA Agonists
  • Muscimol

Grants and funding

This work was supported by grants from the National Natural Science Foundation of China (No. 81171249). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.