Autosomal-dominant Leber Congenital Amaurosis Caused by a Heterozygous CRX Mutation in a Father and Son

Ophthalmic Genet. 2015 Jun;36(2):156-9. doi: 10.3109/13816810.2013.838273. Epub 2013 Oct 4.

Abstract

Background: Leber congenital amaurosis (LCA) is most often an autosomal recessive disorder. We report a father and son with autosomal dominant LCA due to a mutation in the CRX gene.

Materials and methods: DNA screening using an allele specific assay of 90 of the most common LCA-causing variations in the coding sequences of AIPL1, CEP290, CRB1, CRX, GUCY2D, RDH12 and RPE65 was performed on the father. Automated DNA sequencing of his son examining exon 3 of the CRX gene was subsequently performed.

Results: Both father and son have a heterozygous single base pair deletion of an adenine at codon 153 in the coding sequence of the CRX gene resulting in a frameshift mutation.

Conclusion: Mutations involving the CRX gene may demonstrate an autosomal dominant inheritance pattern for LCA.

Keywords: CRX; Leber congenital amaurosis; heterozygous mutation; isoelectric electroretinogram; retinal dystrophy.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • DNA Mutational Analysis
  • Exons / genetics
  • Fathers
  • Genes, Dominant*
  • Genotype
  • Homeodomain Proteins / genetics*
  • Humans
  • Infant
  • Leber Congenital Amaurosis / diagnosis
  • Leber Congenital Amaurosis / genetics*
  • Male
  • Mutation*
  • Nuclear Family
  • Pedigree
  • Sequence Analysis, DNA
  • Trans-Activators / genetics*

Substances

  • Homeodomain Proteins
  • Trans-Activators
  • cone rod homeobox protein