Increased radiosensitivity and radiation-induced apoptosis in SRC-3 knockout mice

J Radiat Res. 2014 May;55(3):443-50. doi: 10.1093/jrr/rrt132. Epub 2013 Dec 5.

Abstract

Steroid receptor coactivator-3 (SRC-3), a multifunctional transcriptional coactivator, plays an important role in regulation of cell apoptosis in chemoresistant cancer cells. However, its role in radiation-induced apoptosis in hematopoietic cells is still unclear. In this study, we used SRC-3 knockout (SRC-3(-/-)) mice to assess the role of SRC-3 in radiation-induced hematopoietic injury in vivo. After a range of doses of irradiation, SRC-3(-/-) mice exhibited lower counts of peripheral blood cells and bone marrow (BM) mononuclear cells and excessive BM depression, which resulted in a significantly higher mortality compared with wildtype mice. Moreover, BM mononuclear cells obtained from SRC-3(-/-) mice showed a remarkable increase in radiation-induced apoptosis. Collectively, our data demonstrate that SRC-3 plays a role in radiation-induced apoptosis of BM hematopoietic cells. Regulation of SRC-3 might influence the radiosensitivity of hematopoietic cells, which highlights a potential therapeutic target for radiation-induced hematopoietic injury.

Keywords: Radiosensitivity; SRC-3; apoptosis; hematopoiesis; knockout mice.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis / radiation effects*
  • Cells, Cultured
  • Leukocytes, Mononuclear / metabolism*
  • Leukocytes, Mononuclear / pathology*
  • Leukocytes, Mononuclear / radiation effects
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Nuclear Receptor Coactivator 3 / genetics
  • Nuclear Receptor Coactivator 3 / metabolism*
  • Radiation Dosage
  • Radiation Injuries, Experimental / metabolism*
  • Radiation Injuries, Experimental / pathology*
  • Radiation Tolerance*
  • Whole-Body Irradiation

Substances

  • Ncoa3 protein, mouse
  • Nuclear Receptor Coactivator 3