Abstract
Six influenza A(H1N1)pdm09 viruses were detected in Sapporo, Japan, between November and December 2013. All six viruses possessed an H275Y substitution in the neuraminidase protein, which confers cross-resistance to oseltamivir and peramivir. No epidemiological link among the six cases could be identified; none of them had received neuraminidase inhibitors before specimen collection. The haemagglutinin and neuraminidase genes of the six viruses were closely related to one another, suggesting clonal spread of a single resistant virus.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Acids, Carbocyclic
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Antiviral Agents / pharmacology*
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Antiviral Agents / therapeutic use
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Child
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Child, Preschool
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Cyclopentanes / pharmacology*
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Cyclopentanes / therapeutic use
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DNA, Viral
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Drug Resistance, Viral
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Female
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Guanidines / pharmacology*
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Guanidines / therapeutic use
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Humans
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Infant
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Influenza A Virus, H1N1 Subtype / classification
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Influenza A Virus, H1N1 Subtype / drug effects*
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Influenza A Virus, H1N1 Subtype / genetics
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Influenza A Virus, H1N1 Subtype / isolation & purification*
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Influenza, Human / drug therapy*
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Influenza, Human / virology
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Japan / epidemiology
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Male
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Microbial Sensitivity Tests
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Molecular Sequence Data
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Mutation
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Neuraminidase / genetics
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Neuraminidase / therapeutic use
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Oseltamivir / pharmacology*
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Oseltamivir / therapeutic use
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Phylogeny
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Sequence Analysis, DNA
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Treatment Outcome
Substances
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Acids, Carbocyclic
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Antiviral Agents
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Cyclopentanes
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DNA, Viral
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Guanidines
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Oseltamivir
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Neuraminidase
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peramivir