Abstract
Structure-based drug design was used to guide the optimization of a series of selective BTK inhibitors as potential treatments for Rheumatoid arthritis. Highlights include the introduction of a benzyl alcohol group and a fluorine substitution, each of which resulted in over 10-fold increase in activity. Concurrent optimization of drug-like properties led to compound 1 (RN486) ( J. Pharmacol. Exp. Ther. 2012 , 341 , 90 ), which was selected for advanced preclinical characterization based on its favorable properties.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Agammaglobulinaemia Tyrosine Kinase
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Arthritis, Rheumatoid / drug therapy*
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Crystallography, X-Ray
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Drug Design
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Humans
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Isoquinolines / chemistry
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Isoquinolines / metabolism
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Isoquinolines / pharmacology*
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Models, Chemical
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Models, Molecular
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Molecular Structure
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Protein Binding
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Protein Structure, Secondary
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Protein Structure, Tertiary
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Protein-Tyrosine Kinases / antagonists & inhibitors*
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Protein-Tyrosine Kinases / chemistry
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Protein-Tyrosine Kinases / metabolism
Substances
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Isoquinolines
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RN486
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Protein-Tyrosine Kinases
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Agammaglobulinaemia Tyrosine Kinase
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BTK protein, human