Targeted next-generation sequencing at copy-number breakpoints for personalized analysis of rearranged ends in solid tumors

PLoS One. 2014 Jun 17;9(6):e100089. doi: 10.1371/journal.pone.0100089. eCollection 2014.

Abstract

Background: The concept of the utilization of rearranged ends for development of personalized biomarkers has attracted much attention owing to its clinical applicability. Although targeted next-generation sequencing (NGS) for recurrent rearrangements has been successful in hematologic malignancies, its application to solid tumors is problematic due to the paucity of recurrent translocations. However, copy-number breakpoints (CNBs), which are abundant in solid tumors, can be utilized for identification of rearranged ends.

Method: As a proof of concept, we performed targeted next-generation sequencing at copy-number breakpoints (TNGS-CNB) in nine colon cancer cases including seven primary cancers and two cell lines, COLO205 and SW620. For deduction of CNBs, we developed a novel competitive single-nucleotide polymorphism (cSNP) microarray method entailing CNB-region refinement by competitor DNA.

Result: Using TNGS-CNB, 19 specific rearrangements out of 91 CNBs (20.9%) were identified, and two polymerase chain reaction (PCR)-amplifiable rearrangements were obtained in six cases (66.7%). And significantly, TNGS-CNB, with its high positive identification rate (82.6%) of PCR-amplifiable rearrangements at candidate sites (19/23), just from filtering of aligned sequences, requires little effort for validation.

Conclusion: Our results indicate that TNGS-CNB, with its utility for identification of rearrangements in solid tumors, can be successfully applied in the clinical laboratory for cancer-relapse and therapy-response monitoring.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Chromosome Breakpoints*
  • Colonic Neoplasms / genetics*
  • DNA Copy Number Variations*
  • High-Throughput Nucleotide Sequencing / methods*
  • Humans
  • Sequence Analysis, DNA / methods*

Grants and funding

This work was supported by a grant to K.-M.H. from the Basic Research Program through the National Research Foundation of Korea (NRF), funded by the Ministry of Education, Science and Technology (1031020), and by a grant to K.-M.H. from the National Cancer Center through the Korean Ministry of Health and Welfare (1110130). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.