Transgenic HLA-DR alpha faithfully reconstitutes IE-controlled immune functions and induces cross-tolerance to E alpha in E alpha 0 mutant mice

Cell. 1989 Aug 11;58(3):583-94. doi: 10.1016/0092-8674(89)90439-x.

Abstract

We have constructed transgenic mice that express the human class II MHC molecule HLA-DR alpha on a genetic background in which the equivalent endogenous gene, H-2 IE alpha, is not expressed. In these mice, DR alpha complemented the E beta chain such that tissue-specific expression of an interspecies hybrid DR alpha-E beta heterodimer was obtained. Despite 25% amino acid differences between DR alpha and E alpha, immune responsiveness to IE-controlled antigens, clonal deletion of IE-reactive T cells, and alloantigenicity were quantitatively and qualitatively indistinguishable in IE-positive mice and in mice that had integrated at least four copies of the transgene. These results demonstrate a remarkable degree of structural, regulatory, and functional conservation. They also suggest that tolerance induction involves only discrete portions of MHC molecules.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Genes
  • Genetic Complementation Test
  • HLA-DR Antigens / genetics*
  • HLA-DR Antigens / immunology
  • Histocompatibility Antigens Class II / genetics*
  • Histocompatibility Antigens Class II / immunology
  • Humans
  • Immunity, Cellular
  • Lymphocyte Culture Test, Mixed
  • Macromolecular Substances
  • Mice
  • Mice, Transgenic
  • Protein Binding
  • Receptors, Antigen, T-Cell / physiology
  • Structure-Activity Relationship
  • T-Lymphocytes / immunology
  • Tissue Distribution

Substances

  • HLA-DR Antigens
  • Histocompatibility Antigens Class II
  • Macromolecular Substances
  • Receptors, Antigen, T-Cell