IL-22, cell regeneration and autoimmunity

Cytokine. 2015 Jul;74(1):35-42. doi: 10.1016/j.cyto.2014.09.007. Epub 2014 Nov 29.

Abstract

IL-22 as a cytokine is described with opposing pro-inflammatory and anti-inflammatory functions. Cell regeneration, tissue remodelling and balance between commensal bacteria in the gut and host immune system are considered as anti-inflammatory features of IL-22, whereas production of IL-22 from Th17 cells links this cytokine to pro-inflammatory pathways. Th17 cells and group 3 innate lymphoid cells (ILC3) are two major producers of IL-22 and both cell types express ROR-γt and Aryl hydrocarbon receptor (AhR) transcription factors. Typically, the immune system cells are the main producers of IL-22. However, targets of this cytokine are mostly non-hematopoietic cells such as hepatocytes, keratinocytes, and epithelial cells of lung and intestine. Association of IL-22 with other cytokines or transcription factors in different cell types might explain its contrasting role in health and disease. In this review we discuss the regulation of IL-22 production by AhR- and IL-23-driven pathways. A clear understanding of the biology of IL-22 will provide new opportunities for its application to improve human health involving many debilitating conditions.

Keywords: Aryl hydrocarbon receptor (AhR); Autoimmune diseases; IL-22; IL-23; Regenerating (Reg) genes.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Autoimmunity*
  • Cell Differentiation
  • Cytokines / metabolism
  • Epithelial Cells / physiology*
  • Gene Expression Regulation
  • Humans
  • Interleukin-17 / metabolism
  • Interleukin-22
  • Interleukin-23 / metabolism*
  • Interleukins / biosynthesis
  • Interleukins / immunology*
  • Interleukins / metabolism*
  • Mice
  • Nuclear Receptor Subfamily 1, Group F, Member 3 / metabolism
  • Receptors, Aryl Hydrocarbon / metabolism*
  • Regeneration
  • Th17 Cells / immunology

Substances

  • Cytokines
  • Interleukin-17
  • Interleukin-23
  • Interleukins
  • Nuclear Receptor Subfamily 1, Group F, Member 3
  • Receptors, Aryl Hydrocarbon