Progenitor cell expansion and impaired hepatocyte regeneration in explanted livers from alcoholic hepatitis

Gut. 2015 Dec;64(12):1949-60. doi: 10.1136/gutjnl-2014-308410. Epub 2015 Mar 2.

Abstract

Objective: In alcoholic hepatitis (AH), development of targeted therapies is crucial and requires improved knowledge of cellular and molecular drivers in liver dysfunction. The unique opportunity of using explanted livers from patients with AH having undergone salvage liver transplantation allowed to perform more in-depth molecular translational studies.

Design: We studied liver explants from patients with AH submitted to salvage transplantation (n=16), from patients with alcoholic cirrhosis without AH (n=12) and fragments of normal livers (n=16). Hepatic cytokine content was quantified. Hepatocyte function and proliferation and the presence of hepatic progenitor cells (HPCs) were evaluated by immunohistochemistry, western blot or quantitative PCR. Mitochondrial morphology was evaluated by electron microscopy.

Results: Livers from patients with AH showed decreased cytokine levels involved in liver regeneration (tumour necrosis factor α and interleukin-6), as well as a virtual absence of markers of hepatocyte proliferation compared with alcoholic cirrhosis and normal livers. Electron microscopy revealed obvious mitochondrial abnormalities in AH hepatocytes. Importantly, livers from patients with AH showed substantial accumulation of HPCs that, unexpectedly, differentiate only into biliary cells. AH livers predominantly express laminin (extracellular matrix protein favouring cholangiocyte differentiation); consequently, HPC expansion is inefficient at yielding mature hepatocytes.

Conclusions: AH not responding to medical therapy is associated with lack of expression of cytokines involved in liver regeneration and profound mitochondrial damage along with lack of proliferative hepatocytes. Expansion of HPCs is inefficient to yield mature hepatocytes. Manoeuvres aimed at promoting differentiation of HPCs into mature hepatocytes should be tested in AH.

Keywords: ALCOHOLIC LIVER DISEASE; CYTOKERATINS; EXTRACELLULAR MATRIX; HEPATOCYTE; LIVER REGENERATION.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Differentiation*
  • Cell Proliferation
  • Cytokine TWEAK
  • DNA Damage
  • DNA, Mitochondrial
  • Hepatitis, Alcoholic / metabolism*
  • Hepatitis, Alcoholic / pathology
  • Hepatocytes / physiology
  • Hepatocytes / ultrastructure
  • Humans
  • Interleukin-6 / metabolism
  • Keratin-7 / metabolism
  • Ki-67 Antigen / metabolism
  • Laminin / metabolism
  • Liver / cytology
  • Liver / metabolism*
  • Liver Cirrhosis, Alcoholic / metabolism*
  • Liver Cirrhosis, Alcoholic / pathology
  • Liver Regeneration / physiology*
  • Mitochondria / ultrastructure
  • NF-kappa B / metabolism
  • Signal Transduction
  • Stem Cells / physiology*
  • Tumor Necrosis Factor-alpha / metabolism
  • Tumor Necrosis Factors / metabolism

Substances

  • Cytokine TWEAK
  • DNA, Mitochondrial
  • Interleukin-6
  • Keratin-7
  • Ki-67 Antigen
  • Laminin
  • NF-kappa B
  • TNFSF12 protein, human
  • Tumor Necrosis Factor-alpha
  • Tumor Necrosis Factors