Involvement of TLR2-MyD88 in abnormal expression of miR-146a in peripheral blood monocytes of patients with chronic hepatitis C

J Huazhong Univ Sci Technolog Med Sci. 2015 Apr;35(2):219-224. doi: 10.1007/s11596-015-1414-5. Epub 2015 Apr 16.

Abstract

miR-146a is an immunoregulatory microRNA closely associated with viral infection. This study investigated the expression changes of miR-146a in peripheral blood monocytes of HCV-infected patients and the mechanism by which the THP-1 cells were stimulated with HCV core protein in vitro. It was found that in the peripheral blood monocytes of HCV-infected patients, miR-146a expression was upregulated. After treated by interferon/ribavirin, miR-146a expression was decreased when HCV RNA became undetectable. HCV core could directly stimulate THP-1 cells to produce miR-146a. Silencing TLR2 and MyD88 could significantly inhibit the expression of miR-146a. It was concluded that the expression of miR-146a in peripheral blood monocytes of HCV-infected patients was abnormally increased. The TLR2-MyD88 signaling pathway may take part in the overexpression of miR-146a in monocytes stimulated with HCV core protein.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Base Sequence
  • Cell Line
  • DNA Primers
  • Female
  • Hepatitis C, Chronic / blood*
  • Humans
  • Male
  • MicroRNAs / blood*
  • Middle Aged
  • Monocytes / metabolism*
  • Myeloid Differentiation Factor 88 / physiology*
  • Reverse Transcriptase Polymerase Chain Reaction
  • Toll-Like Receptor 2 / physiology*
  • Young Adult

Substances

  • DNA Primers
  • MIRN146 microRNA, human
  • MYD88 protein, human
  • MicroRNAs
  • Myeloid Differentiation Factor 88
  • TLR2 protein, human
  • Toll-Like Receptor 2