Nuclear Receptor Coregulators in Metabolism and Disease

Handb Exp Pharmacol. 2016:233:95-135. doi: 10.1007/164_2015_5.

Abstract

Within the past two decades, coregulators have emerged as essential chromatin components of metabolic signaling by nuclear receptors and additional metabolite-sensing transcription factors. Intriguingly, coregulators themselves are efficient sensors and effectors of metabolic stimuli that modulate gene expression at different levels, often via post-translational modifications of histones or other factors. There is already evidence that alterations of expression or function of coregulators contributes to metabolic disease by propagating disease-specific epigenomes linked to the dysregulation of transcription and downstream pathways. In this chapter we review the current progress made in understanding the role of coregulators in metabolic pathways, with a particular emphasis on their study in vivo and in the context of metabolic disease.

Keywords: Coregulators; Inflammation; Metabolic disease; Metabolism; Nuclear receptors.

Publication types

  • Review

MeSH terms

  • Adipose Tissue / metabolism
  • Animals
  • Energy Metabolism
  • Humans
  • Insulin Resistance
  • Metabolic Diseases / etiology*
  • Muscle, Skeletal / metabolism
  • Organ Specificity
  • Protein Processing, Post-Translational
  • Receptors, Cytoplasmic and Nuclear / physiology*
  • Signal Transduction

Substances

  • Receptors, Cytoplasmic and Nuclear