[Regulatory T cells in the treatment of autoimmune myositis in mice: efficacy and mechanism]

Nan Fang Yi Ke Da Xue Xue Bao. 2015 Apr;35(4):602-5.
[Article in Chinese]

Abstract

Objective: To investigate effect of CD4(+) CD25(+) Foxp3(+) Tregs in the treatment of autoimmune myositis (EAM) in mice and explore the possible mechanisms.

Methods: Mouse models of EAM were divided randomly into model group and treatment group, and the latter received infusion of CD4(+) CD25(+) Foxp3(+) Tregs separated from normal mouse spleen by magnetic activated cell sorting. The changes of muscle pathology was observed, and the expression of PD-1 and CTLA-4 in spleen CD4(+) CD25(+) Foxp3(+) Tregs was analyzed using flow cytometry; peripheral blood IL-10 and TGF-β levels were tested using double antibody sandwich ELISA.

Results: Compare with the model group, the mice in the treatment group showed significantly reduced muscular inflammatory cell infiltration, increased blood levels of IL-10 and TGF-β (P<0.05), and increased expression of PD-1 and CTLA-4 in spleen CD4(+) CD25(+) Foxp3(+) Tregs (P<0.05).

Conclusion: CD4(+) CD25(+) Foxp3(+) Tregs reinfusion produces therapeutic effect in mice with EAM by increasing peripheral blood IL-10 and TGF-β levels and PD-1 and CTLA-4 expressions in spleen CD4(+) CD25(+) Foxp3(+) Tregs.

MeSH terms

  • Animals
  • Autoimmune Diseases / immunology*
  • CTLA-4 Antigen / metabolism
  • Cell Separation
  • Cell- and Tissue-Based Therapy
  • Disease Models, Animal
  • Flow Cytometry
  • Interleukin-10 / blood
  • Mice
  • Myositis / immunology*
  • Programmed Cell Death 1 Receptor / metabolism
  • Spleen / immunology
  • T-Lymphocytes, Regulatory / immunology*
  • Transforming Growth Factor beta1 / blood

Substances

  • CTLA-4 Antigen
  • Ctla4 protein, mouse
  • IL10 protein, mouse
  • Pdcd1 protein, mouse
  • Programmed Cell Death 1 Receptor
  • Tgfb1 protein, mouse
  • Transforming Growth Factor beta1
  • Interleukin-10