miR-367 promotes proliferation and stem-like traits in medulloblastoma cells

Cancer Sci. 2015 Sep;106(9):1188-95. doi: 10.1111/cas.12733. Epub 2015 Aug 6.

Abstract

In medulloblastoma, abnormal expression of pluripotency factors such as LIN28 and OCT4 has been correlated with poor patient survival. The miR-302/367 cluster has also been shown to control self-renewal and pluripotency in human embryonic stem cells and induced pluripotent stem cells, but there is limited, mostly correlational, information about these pluripotency-related miRNA in cancer. We evaluated whether aberrant expression of such miRNA could affect tumor cell behavior and stem-like traits, thereby contributing to the aggressiveness of medulloblastoma cells. Basal expression of primary and mature forms of miR-367 were detected in four human medulloblastoma cell lines and expression of the latter was found to be upregulated upon enforced expression of OCT4A. Transient overexpression of miR-367 significantly enhanced tumor features typically correlated with poor prognosis; namely, cell proliferation, 3-D tumor spheroid cell invasion and the ability to generate neurosphere-like structures enriched in CD133 expressing cells. A concurrent downregulation of the miR-367 cancer-related targets RYR3, ITGAV and RAB23, was also detected in miR-367-overexpressing cells. Overall, these findings support the pro-oncogenic activity of miR-367 in medulloblastoma and reveal a possible mechanism contributing to tumor aggressiveness, which could be further explored to improve patient stratification and treatment of this important type of pediatric brain cancer.

Keywords: Cancer stem cell; medulloblastoma; miR-367; microRNA; pluripotency.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • AC133 Antigen
  • Antigens, CD / genetics
  • Brain Neoplasms / genetics*
  • Brain Neoplasms / pathology*
  • Cell Line, Tumor
  • Cell Proliferation / genetics*
  • Down-Regulation / genetics
  • Glycoproteins / genetics
  • Humans
  • Medulloblastoma / genetics*
  • Medulloblastoma / pathology*
  • MicroRNAs / genetics*
  • Octamer Transcription Factor-3 / genetics
  • Peptides / genetics
  • Ryanodine Receptor Calcium Release Channel / genetics
  • Spheroids, Cellular / pathology
  • Up-Regulation / genetics
  • rab GTP-Binding Proteins / genetics

Substances

  • AC133 Antigen
  • Antigens, CD
  • Glycoproteins
  • MIRN367 microRNA, human
  • MicroRNAs
  • Octamer Transcription Factor-3
  • POU5F1 protein, human
  • PROM1 protein, human
  • Peptides
  • Ryanodine Receptor Calcium Release Channel
  • RAB23 protein, human
  • rab GTP-Binding Proteins