CD73-adenosine: a next-generation target in immuno-oncology

Immunotherapy. 2016 Feb;8(2):145-63. doi: 10.2217/imt.15.106. Epub 2016 Jan 25.

Abstract

Cancer immunotherapy has entered in a new era with the development of first-generation immune checkpoint inhibitors targeting the PD1/PD-L1 and CTLA-4 pathways. In this context, considerable research effort is being deployed to find the next generation of cancer immunotherapeutics. The CD73-adenosine axis constitutes one of the most promising pathways in immuno-oncology. We and others have demonstrated the immunosuppressive role of CD73-adenosine in cancer and established proof-of-concept that the targeted blockade of CD73 or adenosine receptors could effectively promote anti-tumor immunity and enhance the activity of first-generation immune checkpoint blockers. With Phase I clinical trials now underway evaluating anti-CD73 or anti-A2A therapies in cancer patients, we here discuss the fundamental, preclinical and clinical findings related to the role of the CD73-adenosinergic pathway in tumor immunity.

Keywords: CD73; adenosine; adenosine receptors; anti-tumor immunity; immune checkpoint.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • 5'-Nucleotidase / antagonists & inhibitors*
  • 5'-Nucleotidase / immunology
  • Adenosine / immunology*
  • Adenosine A2 Receptor Antagonists / therapeutic use*
  • Animals
  • Clinical Trials, Phase I as Topic
  • GPI-Linked Proteins / antagonists & inhibitors
  • GPI-Linked Proteins / immunology
  • Humans
  • Neoplasms / drug therapy*
  • Neoplasms / immunology
  • Neoplasms / pathology
  • Receptor, Adenosine A2A / immunology*

Substances

  • Adenosine A2 Receptor Antagonists
  • GPI-Linked Proteins
  • Receptor, Adenosine A2A
  • 5'-Nucleotidase
  • NT5E protein, human
  • Adenosine