Immunological mechanisms involved in probiotic-mediated protection against Citrobacter rodentium-induced colitis

Benef Microbes. 2016 Jun;7(3):397-407. doi: 10.3920/BM2015.0119. Epub 2016 Mar 1.

Abstract

Inflammatory bowel disease is a group of chronic, incurable inflammatory disorders of the gastrointestinal tract that cause severe diarrhoea, intestinal inflammation, pain, fatigue and weight loss. In this study, we first developed a model of Citrobacter rodentium-induced colitis and then evaluated the protective effects of selected probiotics on inflammation. The results showed that administration of a combination of probiotics including Lactobacillus rhamnosus ATCC 53103, Lactobacillus acidophilus ATCC 4356 and Lactobacillus plantarum A significantly increased the production of CD11c(+) dendritic cells in the spleen (3.62% vs phosphate buffered saline (PBS)-treated control, P<0.01) and mesenteric lymph nodes (MLNs). In addition, the presence of probiotics significantly up-regulated the development of CD4(+)/CD25(+)/Foxp3(+) regulatory T cells in MLNs by approximately 2.07% compared to the effect observed in the PBS-treated control (P<0.01) and down-regulated the expression of inflammatory cytokines, including interleukin-17, tumour necrosis factor-α and interferon-γ, by 0.11, 0.11 and 0.15%, respectively, compared to the effect observed in the PBS-treated control (P<0.01).These effects conferred protection against colitis, as shown by histopathological analyses.

Keywords: Citrobacter rodentium; colitis; dendritic cells; probiotics; regulatory T cells.

MeSH terms

  • Citrobacter rodentium / pathogenicity*
  • Colitis / pathology*
  • Cytokines / metabolism
  • Dendritic Cells / immunology
  • Immune Tolerance
  • Immunologic Factors / administration & dosage*
  • Lactobacillus / immunology*
  • Lymph Nodes / immunology
  • Probiotics / administration & dosage*
  • Spleen / immunology
  • T-Lymphocytes, Regulatory / immunology

Substances

  • Cytokines
  • Immunologic Factors