Interleukin-21 expression in hippocampal astrocytes is enhanced following kainic acid-induced seizures

Neurol Res. 2016 Feb;38(2):151-7. doi: 10.1080/01616412.2015.1135557.

Abstract

Objective: Interleukin-21 (IL-21) is a cytokine that is an important modulator of immune responses. However, its roles in epilepsy are not completely clear. Here, we investigated the expression and distribution of IL-21 in a kainic acid (KA)-induced acute seizure mouse model.

Materials and methods: Mice (n = 146) were randomly divided into an age-matched group, PBS injection group, and a KA injection group. The KA-injected mice were evaluated at 1, 3, and 24 h post-injection. IL-21 mRNA and protein expression levels were measured using RT-PCR and western blotting. Immunohistochemistry and immunofluorescence staining were performed to further characterize the pattern and distribution of IL-21 expression.

Results: The IL-21 mRNA and protein expression levels in the hippocampal tissues of the KA-treated mice were significantly increased as early as 1 h compared with the age-matched mice and PBS-treated mice. After this time point, the expression was reduced, but it remained higher than the level in the PBS-treated mice (p < 0.01). Immunohistochemical staining showed that IL-21 expression was distributed throughout the hippocampus, including areas CA1 and CA3, the dentate gyrus and the hilus. Moreover, immunofluorescence further showed that in the hippocampi of the KA-treated mice, IL-21 was mainly expressed in GFAP-positive astrocytes rather than in NeuN-positive neurons or CD11b-positive microglia.

Significance: Our data suggest that an increase in astrocyte-derived IL-21 expression in hippocampal subregions following KA-induced seizures may have potent regulatory effects on epileptogenesis.

Keywords: Astrocyte; Inflammatory cytokines; Interleukin-21; Kainic acid; Seizures.

MeSH terms

  • Analysis of Variance
  • Animals
  • Astrocytes / drug effects
  • Astrocytes / metabolism*
  • CD11b Antigen / metabolism
  • Disease Models, Animal
  • Excitatory Amino Acid Agonists / toxicity*
  • Gene Expression Regulation / drug effects*
  • Glial Fibrillary Acidic Protein / metabolism
  • Hippocampus / pathology*
  • Interleukins / genetics
  • Interleukins / metabolism*
  • Kainic Acid / toxicity*
  • Mice
  • Mice, Inbred C57BL
  • Phosphopyruvate Hydratase / metabolism
  • RNA, Messenger / metabolism
  • Seizures / chemically induced*
  • Seizures / pathology
  • Time Factors

Substances

  • CD11b Antigen
  • Excitatory Amino Acid Agonists
  • Glial Fibrillary Acidic Protein
  • Interleukins
  • RNA, Messenger
  • Phosphopyruvate Hydratase
  • interleukin-21
  • Kainic Acid