High Throughput Combinatorial Formatting of PcrV Nanobodies for Efficient Potency Improvement

J Biol Chem. 2016 Jul 15;291(29):15243-55. doi: 10.1074/jbc.M115.684241. Epub 2016 May 20.

Abstract

Improving potencies through concomitant blockage of multiple epitopes and avid binding by fusing multiple (different) monovalent Nanobody building blocks via linker sequences into one multivalent polypeptide chain is an elegant alternative to affinity maturation. We explored a large and random formatting library of bivalent (combinations of two identical) and biparatopic (combinations of two different) Nanobodies for functional blockade of Pseudomonas aeruginosa PcrV. PcrV is an essential part of the P. aeruginosa type III secretion system (T3SS), and its oligomeric nature allows for multiple complex binding and blocking options. The library screening yielded a large number of promising biparatopic lead candidates, revealing significant (and non-trivial) preferences in terms of Nanobody building block and epitope bin combinations and orientations. Excellent potencies were confirmed upon further characterization in two different P. aeruginosa T3SS-mediated cytotoxicity assays. Three biparatopic Nanobodies were evaluated in a lethal mouse P. aeruginosa challenge pneumonia model, conferring 100% survival upon prophylactic administration and reducing lung P. aeruginosa burden by up to 2 logs. At very low doses, they protected the mice from P. aeruginosa infection-related changes in lung histology, myeloperoxidase production, and lung weight. Importantly, the most potent Nanobody still conferred protection after therapeutic administration up to 24 h post-infection. The concept of screening such formatting libraries for potency improvement is applicable to other targets and biological therapeutic platforms.

Keywords: PcrV; Pseudomonas aeruginosa (P. aeruginosa); avidity; biparatopic; bivalent; epitope mapping; formatting; high throughput screening (HTS); single-domain antibody (sdAb, Nanobody); type III secretion system (T3SS).

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antigens, Bacterial / immunology*
  • Bacterial Toxins / immunology*
  • Cell Death
  • Combinatorial Chemistry Techniques / methods*
  • Disease Models, Animal
  • Epitope Mapping
  • Epitopes / chemistry
  • Epitopes / immunology
  • Female
  • High-Throughput Screening Assays / methods*
  • Humans
  • Mice, Inbred C57BL
  • Models, Molecular
  • Pneumonia / immunology
  • Pneumonia / microbiology
  • Pneumonia / pathology
  • Pore Forming Cytotoxic Proteins / immunology*
  • Pseudomonas Infections / immunology
  • Pseudomonas aeruginosa / immunology
  • Single-Domain Antibodies / immunology*
  • Vaccine Potency*

Substances

  • Antigens, Bacterial
  • Bacterial Toxins
  • Epitopes
  • Pore Forming Cytotoxic Proteins
  • Single-Domain Antibodies
  • antigen V, Pseudomonas

Associated data

  • PDB/1R6F
  • PDB/2CA5
  • PDB/2V6L