Damaged replication forks tolerate USP7 to maintain genome stability

Mol Cell Oncol. 2015 Jul 6;3(1):e1063571. doi: 10.1080/23723556.2015.1063571. eCollection 2016 Jan.

Abstract

RAD18 functions to promote DNA damage tolerance (DTT), a process that ensures faithful genome duplication. Protein ubiquitylation/deubiquitylation is a critical regulatory mechanism controlling DTT. Recently, we have identified the deubiquitylating enzyme USP7 as a component of the DTT machinery that acts to protect RAD18 from proteasome-dependent degradation.