The interleukin-6-hepcidin-hemoglobin circuit in systemic lupus erythematosus flares

Lupus. 2017 Feb;26(2):200-203. doi: 10.1177/0961203316659153. Epub 2016 Jul 20.

Abstract

Objective Hepcidin regulates iron availability and may be responsible for the anemia of chronic disease because it is induced by interleukin-6. This study investigated the IL-6-hepcidin-hemoglobin axis in patients with lupus. Methods IL-6 and hepcidin were measured in serial serum samples collected before, during and after lupus flares by specific ELISAs. Results During renal and non-renal SLE flare cycles IL-6 did not predict hepcidin and hepcidin did not predict hemoglobin. When lupus nephritis patients were in remission, IL-6 and hepcidin were correlated, but hepcidin and hemoglobin were not. Conclusion Hepcidin does not contribute significantly to anemia during active lupus.

Keywords: Hepcidin; anemia; inflammation; lupus nephritis.

Publication types

  • Observational Study

MeSH terms

  • Anemia / blood
  • Anemia / diagnosis
  • Biomarkers / blood
  • Disease Progression
  • Enzyme-Linked Immunosorbent Assay
  • Female
  • Hemoglobins / metabolism*
  • Hepcidins / blood*
  • Humans
  • Interleukin-6 / blood*
  • Lupus Erythematosus, Systemic / blood*
  • Lupus Erythematosus, Systemic / diagnosis
  • Lupus Nephritis / blood*
  • Lupus Nephritis / diagnosis
  • Male
  • Ohio
  • Prospective Studies
  • Time Factors

Substances

  • Biomarkers
  • Hemoglobins
  • Hepcidins
  • IL6 protein, human
  • Interleukin-6