Gender, apolipoprotein E genotype, and mesial temporal atrophy: 2-year follow-up in patients with stable mild cognitive impairment and with progression from mild cognitive impairment to Alzheimer's disease

Neuroradiology. 2016 Nov;58(11):1143-1151. doi: 10.1007/s00234-016-1740-8. Epub 2016 Sep 2.

Abstract

Introduction: This study aimed to examine the relationship between gender, apolipoprotein E (APOE) genotype, and mesial temporal atrophy in mild cognitive impairment (MCI) with and without progression to Alzheimer's disease (AD).

Methods: We evaluated 236 MCI patients with (n = 121) and without (n = 115) AD progression. Longitudinal MRI-based hippocampal volumes (HV) and entorhinal cortex (ERC) thickness were obtained. The Clinical Dementia Rating Sum of Boxes (CDR-SB) score was used to assess disease severity.

Results: We found a significant effect of APOE, gender, and clinical course (stable MCI versus MCI-AD progression) on HV. There was a significant effect of clinical course and APOE, but not gender, on ERC. Baseline HV and APOE4 status predicted MCI-AD progression in women. Baseline ERC and APOE4 status predicted MCI-AD progression in men. There were significant differences in CDR-SB scores between patients with and without MCI-AD progression, but not between males and females, or APOE4 carriers and non-carriers.

Conclusions: HV, but not ERC, is strongly influenced by gender in MCI. The effects of gender and APOE4 on neuroimaging biomarkers have potentially important implications in the prediction of MCI-AD progression and should be taken into account in clinical trials.

Keywords: Apolipoprotein E4; Entorhinal cortex; Hippocampus; MRI; Mild cognitive impairment; Sex differences.

MeSH terms

  • Aged
  • Alzheimer Disease / epidemiology*
  • Alzheimer Disease / genetics*
  • Alzheimer Disease / pathology
  • Apolipoproteins E / genetics*
  • Atrophy / epidemiology
  • Atrophy / pathology
  • Causality
  • Cognitive Dysfunction / epidemiology*
  • Cognitive Dysfunction / genetics*
  • Cognitive Dysfunction / pathology
  • Comorbidity
  • Disease Progression
  • Entorhinal Cortex / pathology*
  • Female
  • Follow-Up Studies
  • Genetic Predisposition to Disease / epidemiology
  • Genetic Predisposition to Disease / genetics
  • Humans
  • Incidence
  • Longitudinal Studies
  • Male
  • Polymorphism, Single Nucleotide / genetics
  • Risk Factors
  • Sex Distribution
  • United States / epidemiology

Substances

  • Apolipoproteins E