Small-molecule WNK inhibition regulates cardiovascular and renal function

Nat Chem Biol. 2016 Nov;12(11):896-898. doi: 10.1038/nchembio.2168. Epub 2016 Sep 5.

Abstract

The With-No-Lysine (K) (WNK) kinases play a critical role in blood pressure regulation and body fluid and electrolyte homeostasis. Herein, we introduce the first orally bioavailable pan-WNK-kinase inhibitor, WNK463, that exploits unique structural features of the WNK kinases for both affinity and kinase selectivity. In rodent models of hypertension, WNK463 affects blood pressure and body fluid and electro-lyte homeostasis, consistent with WNK-kinase-associated physiology and pathophysiology.

MeSH terms

  • Animals
  • Cardiovascular System / drug effects*
  • Cardiovascular System / metabolism
  • Humans
  • Imidazoles / chemistry
  • Imidazoles / pharmacology*
  • Kidney / drug effects*
  • Kidney / metabolism
  • Kidney Function Tests
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • Protein Kinase Inhibitors / chemistry
  • Protein Kinase Inhibitors / pharmacology*
  • Protein Serine-Threonine Kinases / antagonists & inhibitors*
  • Protein Serine-Threonine Kinases / metabolism
  • Pyrrolidines / chemistry
  • Pyrrolidines / pharmacology*
  • Rats
  • Rats, Sprague-Dawley
  • Small Molecule Libraries / chemistry
  • Small Molecule Libraries / pharmacology*

Substances

  • Imidazoles
  • Protein Kinase Inhibitors
  • Pyrrolidines
  • Small Molecule Libraries
  • WNK463
  • Protein Serine-Threonine Kinases

Associated data

  • PubChem-Substance/316894682
  • PubChem-Substance/316894683
  • PubChem-Substance/316894684
  • PubChem-Substance/316894685
  • PubChem-Substance/316894686
  • PubChem-Substance/316894687