Colistin Reduces LPS-Triggered Inflammation in a Human Sepsis Model In Vivo: A Randomized Controlled Trial

Clin Pharmacol Ther. 2017 Jun;101(6):773-781. doi: 10.1002/cpt.582. Epub 2017 Feb 3.

Abstract

The previously described anti-endotoxin effect of colistin has not been investigated in humans yet. We performed a randomized, double-blind, placebo-controlled crossover trial to determine the degree of colistin-driven modulation of inflammatory response in blood of lipopolysaccharide (LPS)-challenged healthy volunteers in a human endotoxemia model. After a single intravenous dose of 2.5 million IU colistin methanesulfonate, interleukin (IL)-6, IL-8, tumor necrosis factor alpha (TNF-α), and IL-1β concentrations as well as other biomarkers of inflammation such as C-reactive protein, differential leukocyte counts, and body temperature were measured up to 24 h postdose. Colistin significantly decreased the inflammatory cytokine response to LPS in blood of healthy volunteers. This effect was most evident for IL-6, IL-8, and TNF-α. This study is the first to confirm the anti-endotoxin effect of colistin in humans in vivo. Further studies might increase our knowledge on the interaction between colistin and the effectors of the immune system.

Publication types

  • Randomized Controlled Trial

MeSH terms

  • Adult
  • Anti-Inflammatory Agents / administration & dosage
  • Anti-Inflammatory Agents / adverse effects
  • Anti-Inflammatory Agents / therapeutic use*
  • Biomarkers / blood
  • Body Temperature Regulation / drug effects
  • C-Reactive Protein / metabolism
  • Colistin / administration & dosage
  • Colistin / adverse effects
  • Colistin / therapeutic use*
  • Cross-Over Studies
  • Cytokines / blood
  • Double-Blind Method
  • Endotoxemia / blood
  • Endotoxemia / chemically induced
  • Endotoxemia / drug therapy*
  • Endotoxemia / physiopathology
  • Healthy Volunteers
  • Humans
  • Inflammation / blood
  • Inflammation / chemically induced
  • Inflammation / physiopathology
  • Inflammation / prevention & control*
  • Inflammation Mediators / blood
  • Infusions, Intravenous
  • Lipopolysaccharides / toxicity*
  • Male
  • Sepsis / blood
  • Sepsis / chemically induced
  • Sepsis / drug therapy*
  • Sepsis / physiopathology
  • Time Factors
  • Treatment Outcome

Substances

  • Anti-Inflammatory Agents
  • Biomarkers
  • Cytokines
  • Inflammation Mediators
  • Lipopolysaccharides
  • C-Reactive Protein
  • Colistin