Copy number variations in DCC/18q and ERBB2/17q are associated with disease-free survival in microsatellite stable colon cancer

Int J Cancer. 2017 Apr 1;140(7):1653-1661. doi: 10.1002/ijc.30584.

Abstract

We conducted a prospective study to assess the prognostic impact of selected copy number variations (CNVs) in Stage II-III microsatellite stable (MSS) colon cancer. A total of 401 patients were included from 01/2004 to 01/2009. The CNVs in 8 selected target genes, DCC/18q, EGFR/7p, TP53/17p, BLK/8p, MYC/8q, APC/5q, ERBB2/17q and STK6/20q, were detected using a quantitative multiplex polymerase chain reaction of short fluorescent fragment (QMPSF) method. The primary end-point was the impact of the CNVs on the 4-year disease-free survival (DFS). The recurrence rate at 4 years was 20.9%, corresponding to 14% Stage II patients versus 31% Stage III patients (p < 0.0001). The 4-year DFS was significantly decreased in patients with a loss at DCC/18q (p = 0.012) and a gain at ERBB2/17q (p = 0.041). The multivariate analysis demonstrated that Stage III, a loss at DCC/18q and a gain at ERBB2/17q were independent factors associated with DFS. A combination of DCC/18q and ERBB2/17q was also associated with relapse, with the hazard ratio increasing from 1 to 2.4 (95% confidence interval (CI), 1.5-4.1) and 3.1 (95% CI, 1.2-8.4) in the presence of 0, 1 or 2 alterations, respectively (p = 0.0013). CNVs in DCC/18q and ERBB2/17q are significantly associated with DFS in Stage II-III MSS colon cancer.

Keywords: chromosomal instability; colon cancer; copy number variation; disease-free survival; microstellite stable.

Publication types

  • Multicenter Study

MeSH terms

  • Aged
  • Carcinoma / genetics*
  • Carcinoma / mortality
  • Carcinoma / pathology
  • Colonic Neoplasms / genetics*
  • Colonic Neoplasms / mortality
  • Colonic Neoplasms / pathology
  • DCC Receptor
  • DNA Copy Number Variations*
  • DNA Mutational Analysis
  • Disease-Free Survival
  • Female
  • Humans
  • Loss of Heterozygosity
  • Male
  • Microsatellite Repeats
  • Middle Aged
  • Neoplasm Metastasis
  • Neoplasm Recurrence, Local / genetics
  • Phenotype
  • Polymerase Chain Reaction
  • Prognosis
  • Proportional Hazards Models
  • Prospective Studies
  • Proto-Oncogene Proteins B-raf / genetics
  • Receptor, ErbB-2 / genetics*
  • Receptors, Cell Surface / genetics*
  • Treatment Outcome
  • Tumor Suppressor Proteins / genetics*

Substances

  • DCC Receptor
  • DCC protein, human
  • Receptors, Cell Surface
  • Tumor Suppressor Proteins
  • ERBB2 protein, human
  • Receptor, ErbB-2
  • BRAF protein, human
  • Proto-Oncogene Proteins B-raf