miR-218 suppresses gastric cancer cell proliferation and invasion via regulation of angiopoietin-2

Exp Ther Med. 2016 Dec;12(6):3837-3842. doi: 10.3892/etm.2016.3893. Epub 2016 Nov 11.

Abstract

Novel targeted therapies need to be developed for gastric cancer, the third most common cancer type and the second most common cause of cancer-related mortality in China. Previous studies indicate that angiopoietin (Ang)-2 serves a role in the proliferation, migration, invasion and adhesion of malignant cells. The present study identified, using functional studies, that exogenous expression of miR-218 increased migration of NCI-87 and HGC-27 gastric cancer cells, which coincided with a reduction in the expression of Ang-2. In addition, intratumoral delivery of miR-218 inhibited proliferation and angiogenesis of gastric cancer cells in vivo, with a corresponding decreased in Ang-2 expression. These results indicate that miR-218 serves an important role in gastric cancer tumorigenesis through regulating the expression of Ang-2. Therefore, components of miR-218/Ang-2 signaling could provide novel therapeutic targets for the treatment of gastric cancer.

Keywords: angiogenesis; angiopoietin-2; gastric cancer; miR-218; proliferation.