FNDC3B is another novel partner fused to RARA in the t(3;17)(q26;q21) variant of acute promyelocytic leukemia

Blood. 2017 May 11;129(19):2705-2709. doi: 10.1182/blood-2017-02-767707. Epub 2017 Mar 17.

Authors

Chi Keung Cheng¹, Angela Z Wang¹, Terry H Y Wong¹, Thomas S K Wan¹, Joyce S Cheung¹, Radha Raghupathy², Natalie P H Chan¹, Margaret H L Ng^{1

3}

Affiliations

¹ Blood Cancer Cytogenetics and Genomics Laboratory, Department of Anatomical and Cellular Pathology, Prince of Wales Hospital.
² Department of Clinical Oncology, Prince of Wales Hospital, and.
³ State Key Laboratory in Oncology in South China, The Chinese University of Hong Kong, Hong Kong SAR, China.

PMID: 28314734
DOI: 10.1182/blood-2017-02-767707

No abstract available

Publication types

Case Reports
Letter

MeSH terms

Adult
Chromosomes, Human, Pair 17 / genetics*
Chromosomes, Human, Pair 17 / metabolism
Chromosomes, Human, Pair 3 / genetics*
Chromosomes, Human, Pair 3 / metabolism
Fibronectins / genetics*
Fibronectins / metabolism
Humans
Leukemia, Promyelocytic, Acute / genetics*
Leukemia, Promyelocytic, Acute / metabolism
Male
Oncogene Proteins, Fusion / genetics*
Oncogene Proteins, Fusion / metabolism
Retinoic Acid Receptor alpha / genetics*
Retinoic Acid Receptor alpha / metabolism
Translocation, Genetic*

Substances

FNDC3B protein, human
Fibronectins
Oncogene Proteins, Fusion
RARA protein, human
Retinoic Acid Receptor alpha