Correspondence: Oncogenic MYC persistently upregulates the molecular clock component REV-ERBα

Nat Commun. 2017 Mar 23:8:14862. doi: 10.1038/ncomms14862.

Authors

Brian J Altman^{1

2

3}, Annie L Hsieh^{1

2

3}, Arvin M Gouw^{1

2

3}, Chi V Dang^{1

2

3}

Affiliations

¹ Abramson Family Cancer Research Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA.
² Abramson Cancer Center, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA.
³ Division of Hematology-Oncology, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA.

No abstract available

Publication types

Letter
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't

MeSH terms

Animals
Cell Line, Tumor
Circadian Clocks / genetics*
Gene Expression Regulation, Neoplastic*
Humans
Liver Neoplasms / genetics
Liver Neoplasms / metabolism
Liver Neoplasms / pathology
Mice
Nuclear Receptor Subfamily 1, Group D, Member 1 / genetics*
Nuclear Receptor Subfamily 1, Group D, Member 1 / metabolism
Proto-Oncogene Proteins c-myc / genetics*
Proto-Oncogene Proteins c-myc / metabolism
Time Factors

Substances

MYC protein, human
NR1D1 protein, human
Nuclear Receptor Subfamily 1, Group D, Member 1
Proto-Oncogene Proteins c-myc

Grants and funding