Differences in the mechanism of induction of interferon-alpha by herpes simplex virus and herpes simplex virus-infected cells

Arch Virol. 1988;103(3-4):219-29. doi: 10.1007/BF01311094.

Abstract

The cellular source of IFN alpha after induction with Herpes simplex virus type-1 (HSV) and HSV-infected fibroblasts was investigated by using human peripheral blood mononuclear cell (PBMC) populations, purified according to conventional procedures, and which included T- and B-lymphocytes as well as monocytes. It appears that the cells responding to HSV virions are monocytes, whereas the PBMC population induced by HSV-infected cells is represented by B-lymphocytes. Furthermore, by using monoclonal antibody (MoAb) to HLA class I and class II products, it appears that different membrane structures are involved in the induction of IFN by HSV virions, as opposed to HSV-infected cells. In fact, most anti-HLA class II MoAbs inhibit IFN induction by HSV-infected cells, and not IFN induction by HSV virions.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antibodies, Monoclonal
  • B-Lymphocytes / metabolism
  • Cell Membrane / physiology
  • Fibroblasts / microbiology*
  • HLA Antigens / immunology
  • Humans
  • Interferon Type I / biosynthesis*
  • Leukocytes, Mononuclear / metabolism*
  • Leukocytes, Mononuclear / microbiology
  • Monocytes / metabolism
  • Simplexvirus / physiology*

Substances

  • Antibodies, Monoclonal
  • HLA Antigens
  • Interferon Type I