Inflammatory Stroke Extracellular Vesicles Induce Macrophage Activation

Stroke. 2017 Aug;48(8):2292-2296. doi: 10.1161/STROKEAHA.117.017236. Epub 2017 May 23.

Abstract

Background and purpose: Extracellular vesicles (EVs) are protein-lipid complexes released from cells, as well as actively exocytosed, as part of normal physiology, but also during pathological processes such as those occurring during a stroke. Our aim was to determine the inflammatory potential of stroke EVs.

Methods: EVs were quantified and analyzed in the sera of patients after an acute stroke (<24 hours; OXVASC [Oxford Vascular Study]). Isolated EV fractions were subjected to untargeted proteomic analysis by liquid chromatography mass-spectrometry/mass-spectrometry and then applied to macrophages in culture to investigate inflammatory gene expression.

Results: EV number, but not size, is significantly increased in stroke patients when compared to age-matched controls. Proteomic analysis reveals an overall increase in acute phase proteins, including C-reactive protein. EV fractions applied to monocyte-differentiated macrophage cultures induced inflammatory gene expression.

Conclusions: Together these data show that EVs from stroke patients are proinflammatory in nature and are capable of inducing inflammation in immune cells.

Keywords: C-reactive protein; acute-phase proteins; extracellular vesicles; macrophages; stroke.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Aged, 80 and over
  • Cohort Studies
  • England / epidemiology
  • Extracellular Vesicles / metabolism*
  • Female
  • Humans
  • Inflammation / blood
  • Inflammation / diagnosis
  • Inflammation / epidemiology
  • Inflammation Mediators / blood*
  • Macrophage Activation / physiology*
  • Macrophages / metabolism*
  • Male
  • Population Surveillance
  • Stroke / blood*
  • Stroke / diagnosis
  • Stroke / epidemiology

Substances

  • Inflammation Mediators